r cell lines (ATCC)
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r cell lines
R Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 180 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/r+cell+lines/MM%2E1R/bio_rxiv__64898__2026__04__04__716518-21-0-6
Average 95 stars, based on 180 article reviews
R Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 180 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/r+cell+lines/MM%2E1R/bio_rxiv__64898__2026__04__04__716518-21-0-6
Average 95 stars, based on 180 article reviews
r cell lines - by Bioz Stars,
2026-09
95/100 stars
Images
1) Product Images from "Defining the RNA Modification Landscape of Multiple Myeloma Reveals METTL3-Dependent m 6 A Regulation of NEAT1"
Article Title: Defining the RNA Modification Landscape of Multiple Myeloma Reveals METTL3-Dependent m 6 A Regulation of NEAT1
Journal: bioRxiv
doi: 10.64898/2026.04.04.716518
Figure Legend Snippet: a. Venn diagram showing overlap of m 6 A-modified lncRNAs identified by direct RNA-seq and meRIP-Seq. b. Read depth of NEAT1 m 6 A sites identified by direct RNA-seq. c. NEAT1 m 6 A site positions identified by both direct RNA-seq (red) and meRIP-Seq (blue). d-f. meRIP RT-qPCR data validating m 6 A site 1611 on NEAT1 in d. RPMI 8226, e. MM.1S, and f. MM.1R relative to negative control IgG. ILF3 mRNA serves as positive control and GAPDH serves as negative control. *fold enrichment > 5, # fold enrichment > 50, n.s. not significant.
Techniques Used: Modification, RNA Sequencing, Quantitative RT-PCR, Negative Control, Positive Control
Figure Legend Snippet: a. UMAP plot of B and plasma single-cell RNA sequencing data for METTL3 (left plot) from healthy (red) and multiple myeloma (MM) patients (blue) (right plot). b. Expression of METTL3 in MM samples compared to healthy samples. c and d . Expression of METTL3 and NEAT1 following small interfering RNA (siRNA)-mediated METTL3 knockdown in c. MM.1S cells and d. RPMI 8226 cells. e. Decreased viability in MM.1S cells following siRNA-mediated METTL3 knockdown, as measured by ApoTox-Glo assay. f. Decreased viability and increased apoptosis in RPMI 8226 cells following siRNA-mediated METTL3 knockdown, as measured by ApoTox-Glo assay. g. Expression of METTL3 and NEAT1 in MM.1R cells with a METTL3 overexpression vector. h. Increased viability and decreased apoptosis in MM.1R cells with a METTL3 overexpression vector, as measured by ApoTox-Glo assay. i. m 6 A blot showing global m 6 A levels in MM cells after 72-hour treatment with METTL3 inhibitor STM2457. j. Decreased viability and increased apoptosis in RPMI 8226 cells with increasing concentrations of STM2457 treatment, as measured by ApoTox-Glo assay k. Decreased viability in RPMI 8226 cells with increasing concentrations of STM2457 treatment, as measured by Celltiter-Glo Assay. *p value < 0.05, **p value < 0.005, ***p value < 0.0005, #p value < 0.00005, n.s. not significant
Techniques Used: Clinical Proteomics, Single Cell, RNA Sequencing, Expressing, Small Interfering RNA, Knockdown, Glo Assay, Over Expression, Plasmid Preparation
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